• 极光加速器官方网站

    Antibodies inhibit virus-induced paralytic disease

    A human monoclonal antibody shows potential to prevent or treat acute flaccid myelitis associated with enterovirus D68 infection

    Kara Jianing Fu/Purdue University

  • Monitoring SARS-CoV-2 exposure

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    STURTI/ISTOCK

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    Signaling by the B cell–activating receptor TACI drives T cell–independent IgA responses to gut commensal bacteria

    Grasset et al./Science Immunology

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    A rare human allele renders MR1 unable to present microbially derived antigen, resulting in a selective loss of MAIT cells

    Howson et al./Science Immunology

  • 极光加速器安卓

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    LAG3 shedding from conventional CD4+ T cells drives responsiveness to anti-PD1 immunotherapy (see the related Focus by Seidel and Bengsch)

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  • TH17 sustenance via IL-6

    Ongoing classic IL-6 receptor signaling is required to maintain the TH17 program, acting upstream of IL-23 to sustain RORγt expression

    Harbour et al./Science Immunology

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    Click on the image to access a special collection that includes all of Science Immunology's COVID-19 papers

    Stanislav Krasilnikov\TASS via Getty Images

Contents

July 2023
Vol 5, Issue 49

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    • SARS-CoV-2 seroprevalence among parturient women in Philadelphia

      By Dustin D. Flannery, 极光加速破解, Miren B. Dhudasia, Sagori Mukhopadhyay, Madeline R. Pfeifer, Emily C. Woodford, Jeffrey S. Gerber, 极光加速器官方网站, Marcus J. Bolton, 极光加速破解, Eileen C. Goodwin, Elizabeth M. Anderson, Allison R. Greenplate, 极光加速器安卓, Nicholas Han, Ajinkya Pattekar, Jeanette Dougherty, Oliva Kuthuru, Divij Mathew, Amy E. Baxter, Laura A. Vella, 极光加速器官方网站, Anurag Verma, Rita Leite, Jeffrey S. Morris, Daniel J. Rader, Michal A. Elovitz, E. John Wherry, Karen M. Puopolo, Scott E. Hensley

      Science Immunology Open Access

      Over six percent of parturient women in Philadelphia (4 April–3 June 2023) had serological evidence of exposure to SARS-CoV-2.

      • Abstract
      • Full Text
      • PDF
      • Supplementary Materials

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      By Daniel M. Altmann, Rosemary J. Boyton

      Science Immunology

      Answers are starting to emerge about target antigens and cytokine profiles of CD4 and CD8 subsets responding to SARS-CoV-2.

      • Abstract
      • Full Text
      • PDF

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    • 极光加速器安卓

      By 极光加速器安卓, M. Betina Pampena, Wenzhao Meng, Aaron M. Rosenfeld, Caroline A.G. Ittner, Ariel R. Weisman, Roseline S. Agyekum, Divij Mathew, 极光加速官网, Laura A. Vella, Oliva Kuthuru, Sokratis A. Apostolidis, Luanne Bershaw, Jeanette Dougherty, Allison R. Greenplate, Ajinkya Pattekar, Justin Kim, Nicholas Han, Sigrid Gouma, Madison E. Weirick, Claudia P. Arevalo, Marcus J. Bolton, Eileen C. Goodwin, Elizabeth M. Anderson, Scott E. Hensley, Tiffanie K. Jones, Nilam S. Mangalmurti, Eline T. Luning Prak, E. John Wherry, Nuala J. Meyer, Michael R. Betts

      Science Immunology Open Access

      Profound plasmablast expansion, innate cell modulation, and T cell activation are defining features of severe COVID-19.

      • Abstract
      • Full Text
      • PDF
      • Supplementary Materials

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    • Shed it, and help—LAG3 cleavage drives conventional CD4+ T cells to overcome resistance to PD-1 immunotherapy

      By Lea Seidel, Bertram Bengsch

      Science Immunology

      LAG3 cleavage from conventional CD4+ T cells, but not CD8+ T cells, is required for effective PD-1 blockade (see the related Research Article by Andrews 极光加速官网.).

      • Abstract
      • Full Text
      • PDF

    • Human effector T cells express TOX—Not so “TOX”ic after all

      By 极光加速官网, Axel Kallies

      Science Immunology

      TOX expression is not restricted to exhausted T cells but is a characteristic of all human effector CD8+ T cells (see the related Research Article by Sekine et al.).

      • Abstract
      • Full Text
      • PDF

  • mogo vpn

    • green极光加速器

      By E. K. Grasset, A. Chorny, S. Casas-Recasens, C. Gutzeit, 极光加速官网, I. Thomsen, L. Chen, Z. He, 极光加速器官方网站, M. A. Oropallo, P. Veeramreddy, M. Uzzan, A. Mortha, J. Carrillo, B. S. Reis, green极光加速器, J. Sintes, G. Magri, P. J. Maglione, C. Cunningham-Rundles, R. J. Bram, green极光加速器, S. Mehandru, O. Pabst, A. Cerutti

      极光加速器官方网站

      Signaling by the B cell–activating receptor TACI drives T cell–independent IgA responses to gut commensal bacteria.

      • Editor's Summary
      • Abstract
      • Full Text
      • PDF
      • green极光加速器

    • Immunophenotyping of COVID-19 and influenza highlights the role of type I interferons in development of severe COVID-19

      By Jeong Seok Lee, Seongwan Park, Hye Won Jeong, Jin Young Ahn, Seong Jin Choi, Hoyoung Lee, Baekgyu Choi, Su Kyung Nam, Moa Sa, Ji-Soo Kwon, Su Jin Jeong, Heung Kyu Lee, Sung Ho Park, Su-Hyung Park, Jun Yong Choi, Sung-Han Kim, Inkyung Jung, Eui-Cheol Shin

      Science Immunology

      Single-cell RNA sequencing of blood immune cells reveals type I interferon–associated hyper-inflammation in severe COVID-19.

      • Editor's Summary
      • Abstract
      • Full Text
      • PDF
      • Supplementary Materials

    • Absence of mucosal-associated invariant T cells in a person with a homozygous point mutation in MR1

      By green极光加速器, Wael Awad, Anouk von Borstel, Hui Jing Lim, Hamish E. G. McWilliam, Maria L. Sandoval-Romero, Shamik Majumdar, Abdul Rezzak Hamzeh, Thomas D. Andrews, David H. McDermott, Philip M. Murphy, 极光加速破解, Jeffrey Y. W. Mak, Ligong Liu, David P. Fairlie, James McCluskey, Jose A. Villadangos, 极光加速器安卓, Stephen J. Turner, Martin S. Davey, Samar Ojaimi, Jamie Rossjohn

      极光加速官网

      A rare human allele renders MR1 unable to present microbially derived antigen, resulting in a selective loss of MAIT cells.

      • Editor's Summary
      • Abstract
      • Full Text
      • PDF
      • Supplementary Materials

    • TH17 cells require ongoing classic IL-6 receptor signaling to retain transcriptional and functional identity

      By green极光加速器, green极光加速器, Steven J. Witte, Carlene L. Zindl, Min Gao, Trenton R. Schoeb, Gareth W. Jones, Simon A. Jones, Robin D. Hatton, Casey T. Weaver

      Science Immunology

      Ongoing classic IL-6 receptor signaling is required to maintain the TH17 program, acting upstream of IL-23 to sustain RORγt expression.

      • Editor's Summary
      • Abstract
      • Full Text
      • PDF
      • 极光加速器官方网站

    • Resistance to PD1 blockade in the absence of metalloprotease-mediated LAG3 shedding

      By 极光加速破解, 极光加速器安卓, Jessica M. Moskovitz, Andrea L. Szymczak-Workman, Chang Liu, Anthony R. Cillo, 极光加速破解, 极光加速官网, Kelly D. Moynihan, Ichiro Taniuchi, Darrell J. Irvine, John M. Kirkwood, 极光加速器安卓, Robert L. Ferris, Tullia C. Bruno, Creg J. Workman, Dario A. A. Vignali

      Science Immunology

      LAG3 shedding from conventional CD4+ T cells drives responsiveness to anti-PD1 immunotherapy (see the related Focus by Seidel and Bengsch).

      • Editor's Summary
      • Abstract
      • Full Text
      • PDF
      • Supplementary Materials

    • The cytoskeletal regulator HEM1 governs B cell development and prevents autoimmunity

      By Elisabeth Salzer, Samaneh Zoghi, Máté G. Kiss, 极光加速官网, Christina Rashkova, 极光加速官网, Matthias Haimel, René Platzer, Michael Caldera, Rico Chandra Ardy, Birgit Hoeger, Jana Block, 极光加速破解, Celine Sin, Sepideh Shahkarami, Renate Kain, Vahid Ziaee, Peter Hammerl, Christoph Bock, Jörg Menche, Loïc Dupré, 极光加速官网, Michael Sixt, 极光加速器安卓, Klemens Rottner, Christoph J. Binder, Theresia E. B. Stradal, Nima Rezaei, Kaan Boztug

      Science Immunology

      HEM1 deficiency causes heritable autoimmunity and immunodeficiency.

      • Editor's Summary
      • 极光加速破解
      • Full Text
      • PDF
      • Supplementary Materials

    • green极光加速器

      By Takuya Sekine, André Perez-Potti, Son Nguyen, Jean-Baptiste Gorin, Vincent H. Wu, Emma Gostick, Sian Llewellyn-Lacey, 极光加速破解, Sara Falck-Jones, Sindhu Vangeti, Meng Yu, Anna Smed-Sörensen, 极光加速官网, Michael Uhlin, Johan K. Sandberg, 极光加速器官方网站, Piotr Nowak, Paul A. Goepfert, David A. Price, Michael R. Betts, Marcus Buggert

      Science Immunology

      The transcription factor TOX is not exclusively linked to T cell exhaustion (see the related Focus by Utzschneider and Kallies).

      • 极光加速官网
      • Abstract
      • Full Text
      • PDF
      • Supplementary Materials

    • green极光加速器

      By 极光加速器安卓, Jianing Fu, Nurgun Kose, Lauren E. Williamson, Robin Bombardi, Ian Setliff, Ivelin S. Georgiev, 极光加速器官方网站, Michael G. Rossmann, Yury A. Bochkov, James E. Gern, Richard J. Kuhn, James E. Crowe, Jr.

      Science Immunology

      A human monoclonal antibody shows potential to prevent or treat acute flaccid myelitis associated with enterovirus D68 infection.

      • Editor's Summary
      • Abstract
      • Full Text
      • PDF
      • Supplementary Materials

  • mogo vpn

    • Human inborn errors of immunity: An expanding universe

      By Luigi D. Notarangelo, Rosa Bacchetta, Jean-Laurent Casanova, Helen C. Su

      green极光加速器

      This Review summarizes recent advances in the molecular, cellular, and clinical characterization of human inborn errors of immunity.

      • Abstract
      • Full Text
      • green极光加速器

  • 极光加速破解

    • Finding Camel-ot: A Holy Grail against pandemic SARS-CoV-2?

      By Deborah Soong, Rachel Leeman, Asha Pillai

      Science Immunology

      Engineered camelid antibody multimers can potently block SARS-CoV-2 viral entry.

      • Abstract
      • Full Text

    • Let’s talk about sex

      By James C. Patti, Gabriel K. Griffin

      Science Immunology

      Complement gene variation drives sex biases in autoimmune disease.

      • Abstract
      • Full Text

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ONLINE COVER Frozen Footprints of Antibody Fabs. This month's cover features a 3D reconstruction of the capsid surface of human enterovirus D68 (EV-D68), a virus linked to an emerging polio-like illness called acute flaccid myelitis (AFM). The blue and gold patches represent the distinct binding sites on the icosahedral capsid (colored in cyan) occupied by two neutralizing human monoclonal antibodies to EV-D68 identified by Vogt 极光加速器安卓. The image was generated by cryo–electron microscopy of immune complexes formed by mixing virions with a 200-fold molar excess of antibody Fab fragments. The antibody depicted in blue is a potential therapeutic agent in human AFM based on its broad reactivity with EV-D68 clades and its ability to protect mice in a model of EV-D68–induced neurologic disease. [CREDIT: KARA JIANING FU/PURDUE UNIVERSITY]